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This neuronal gene activation correlates with observed anti-pain, anti-anxiety, and cognitive enhancement effects in animal studies at doses as low as 0.5 g/kg body weight
Conclusion This study revealed that Mcp1, located at vacuole and mitochondrial contact sites, is essential for the phosphorylation and nuclear localization of the transcription factor Cap1 under oxidative stress and affects the oxidative stress response and hyphal development of C
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