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These new findings encourage a novel working hypothesis for further research, that a chemical-modification mechanism enables transformation of HGF through interaction or reaction with LASSS under physiological conditions, resulting in the retention of HGFs biological activity after exposure to peroxynitrite, at least until an enhanced form of HGF undergoes metabolic turnover in vivo

Under normal physiological conditions, both transcription factors remain in harmony [60]

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The CTR is also expressed at the cell surface independently of RAMPs and has a distinct phenotype, exhibiting potent responses to human, and other species of CT peptides, but weak responses to amylin