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A study highlighted S100A1s ability to restore calcium cycling in cardiomyocytes, improving contractility in heart failure models, and gene therapy using AAV-S100A1 showed reduced fibrosis and hypertrophy in rodents (Rostami et al., 2025)
doi: 10.3390/separations9070165 355 ThomsonCGarciaALEdwardsCA
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The obtained results are typical for old animals (Table 2)
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