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Such a mechanism may explain the peptide's ability to enhance healing without causing excessive or dysregulated tissue growth, as its effects remain dependent on physiological growth factor signaling that is naturally upregulated during injury and repair processes
By inhibiting NF-kB activation, it could potentially reduce the inflammatory cascade that drives flare severity without compromising the immune response to concurrent infections, a common concern during lupus flares when patients may also be on immunosuppressive medications
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