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No significant prolactin elevation occurs, eliminating the gynecomastia concerns associated with GHRP-2 at higher doses
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This is clearly demonstrated by experiments in which the administration of the neurotoxin MPTP, which blocks Complex I of the mitochondrial electron transport chain, induces characteristic features of PD in mice [56, 57]
During the chronic phase, the goal is to increase the metabolic capacity of surviving networks and enable rehabilitation-driven plasticity [29, 41,42,43]
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