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Together, these changes mean that a much higher fraction of administered IGF-1 LR3 reaches tissue receptors as free ligand compared to an equivalent molar dose of native IGF-1

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Phosphatidylethanolamine-binding protein 1 (PEBP1) and arachidonate 15-lipoxygenase (ALOX15) can interfere with phospholipid alcohol synthesis and induce ferroptosis [5, 52]

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