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When combined with GLP-1 agonists, which suppress appetite through an entirely different pathwaydirect stimulation of GLP-1 receptors in the hypothalamuscaffeine does not interfere with or reduce the medication's effectiveness
Activating these receptorsimproves insulin sensitivity, reduces glucagon release, slows stomach emptying,and sends satiety signals to the brain
When these receptors are activated, they help switch off the production of Matrix Metalloproteinases (MMPs)
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