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Lethal hemorrhagic disease and clinical illness associated with elephant endotheliotropic herpesvirus 1 are caused by primary infection: implications for the detection of diagnostic proteins
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Elevated homocysteine levels in the blood can downregulate CSE expression and nitrosylate CSE (CSE-SNO), reducing its catalytic activity and H 2 S production, causing atherosclerosis
The regulatory factors of ferroptosis and other forms of cell death also overlap
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