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Additionally, obesity and IR both contribute to -cell dysfunction and apoptosis, mechanistically explained by glucotoxicity and increased immunogenicity (the accelerator hypothesis) or by -cell exhaustion due to increased metabolic demand (the overload hypothesis) [23]
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The sustained receptor engagement may explain why Retatrutide produces superior weight loss outcomes compared to medications with shorter durations of action, though individual responses vary based on multiple factors including diet, activity levels, and metabolic baseline
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