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Methods In order to gain insights into the molecular pathways underlying progression of emphysema and explore computational strategies for identifying COPD therapeutics, we profiled gene expression in lung tissue samples obtained from regions within the same lung with varying amounts of emphysematous destruction from smokers with COPD (8 regions 8 lungs = 64 samples)
Therefore, to explore how ACTL6A regulates GCLC, we assessed genome-wide ACTL6A binding by performing a chromatin immunoprecipitation high-throughput sequencing (ChIP-seq) analysis of endogenous ACTL6A in GC cells
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PubMed Link Vasiljevic D, et al
