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In endometriotic cells, ferroptosis resistance mechanisms are up-regulated, including increased expression of GPx4 and GSH, and fibulin 1 (FBLN1) which inhibits ferroptosis, thereby promoting cell survival and lesion progression
Topical medications are the first-line therapy and include prostaglandin analogues, -blockers, -agonists, carbonic anhydrase inhibitors, and Rho-kinase inhibitors, each with distinct mechanisms and side-effect profiles [12,13,14]
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