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Ultimately, this FOXO4-DRI-mediated mechanism selectively eliminates senescent endothelial cells, which in turn attenuates vascular inflammation and improves endothelial-dependent vasodilation, conferring a significant protective effect on vascular function

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IGF-1 LR3 has been used in bone biology research, particularly in osteoporosis models and fracture healing studies, where sustained IGF1R activation needs to be maintained throughout the experimental period