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The aggressive biology of PDAC is further compounded by a characteristically dense, desmoplastic stroma that fosters an immunosuppressive TME, contributing to immune evasion and treatment failure [2]

The mechanism and result are similar to that of GSTP and JNK, in that GSTM1 sequesters ASK1 through complex formation and prevents its induction of the pro-apoptotic p38 and JNK portions of the MAPK signaling cascade

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