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score 4: complete infarction (100%)

Conclusion There has been extensive research into the effects of diverse classes of medications targeting different pathophysiological mechanisms including defective contraction or coordination of the stomach manifesting with symptoms and objective retardation of gastric emptying

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Nevertheless, the effect of ethanol consumption on the total hepatic GSH pool is equivocal in that studies have shown decreased, unchanged or increased levels 12,13,14 , most likely related to differences in ethanol exposure regimes and/or analytical assays