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When myocardial damage causes abnormalities in mitochondrial substrate utilization, oxidative phosphorylation, and a decrease in high-energy phosphate compounds, the myocardium undergoes metabolic remodeling, resulting in cardiac energy deprivation and further exacerbating myocardial damage
Additionally, recent clinical evidence has demonstrated that newly diagnosed hypothyroid individuals exhibit elevated malondialdehyde (MDA) and decreased total antioxidant capacity, supporting the link between impaired antioxidant status and heightened oxidative stress (Sultana et al
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