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and GIP receptors have also been demonstrated to activate phosphoinositide 3-kinase (PI3K) and its downstream effector Akt, which modulates mitochondrial function, metabolism, and cell survival
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[2] This increase in MMP-2 was also accompanied by a rise in MMP-2 mRNA levels, indicating that GHK-Cu potentially enhances MMP-2 expression at the transcriptional level
52333003 to X.J.), Basic Research Program of Jiangsu (BK20244003) and the Natural Science Foundation of Jiangsu Province (BK20202002)
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