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The totality of evidence suggests that the effects of DPP-4 inhibitors on the kidney may be primarily mediated through potentiation of SDF-1 rather than of GLP-1
This dual action provides approximately 3x greater fat metabolism through the GIP pathway
The earlier SURPASS trials characterized glycemic parameters in type 2 diabetes cohorts
The absence of established treatment protocols makes it difficult to determine optimal dosing regimens, treatment duration, and comprehensive monitoring requirements
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