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The abnormal release of Ca 2+ from the mitochondria to the cytosol leads to the activation of microglia, neuroinflammation, and finally, cell death [29, 93]
Pathological changes in ON lesions resemble those in chronic inactive MS plaques [163, 164], with each neural lesion exhibiting characteristics of long-term damage
Outside the spaces glass doors there is a giant photo of Cuban, who owns the Mavericks, grimacing in a suit
IRE1XBP1 signaling in leukocytes controls prostaglandin biosynthesis and pain
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