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Our recent work identified multiple RBPs contributing to the differential splicing between glutamatergic and GABAergic neurons in the cortex, with Mbnl2, Celf2 and Khdrbs3 (Slm2) more highly expressed in glutamatergic neurons and facilitating glutamatergic neuron-specific splicing, while Elavl2 and Qk are more highly expressed in GABAergic neurons, facilitating GABAergic neuron-specific splicing 12 (Fig

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Specifically, we found that GSSG elevation suppressed the phosphorylation/activation of IKK-, a proximal kinase in the NF-B signaling pathway that phosphorylates IB-, leading to its proteasomal degradation and subsequent NF-B activation