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These data suggest that ferroptosis plays a critical role in renal fibrosis by sustaining the activation of TGF-/Smad signaling and promoting the release of downstream profibrotic factors, ultimately contributing to fibrogenesis

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Clinical evidence and patient experiences suggest GLP-1 treatments may quiet the relentless mental preoccupation with eating, cravings, and meal planning that characterises food noise

Concurrently, inflamed adipose tissue secretes elevated free fatty acids, pro-inflammatory cytokines, and adipokines that exacerbate hepatic insulin resistance, lipotoxicity, and fibrogenesis [2]