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The results demonstrated that the infusion of GLP-1 during high level of glycemia significantly protects endothelial dysfunction and decreases hyperglycemia-induced oxidative stress see Table 2
Further studies revealed that iNOS is highly expressed in Norway rats ( Figure 1 ), and iNOS interferes with mitochondrial respiration and energy production by producing NO ( S
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both of which contribute to GSH synthesis and neuroimmune modulation (5-6)
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