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In sepsis mouse models pretreated with Gln, the activation of CD4 + T cells was in equilibrium, and the expression of the anti-apoptotic protein Bcl-2 was more pronounced (43)
Here, it functions more fundamentally
The drug developed nearly 15 years ago at Northwestern University, Illinois targets the GSK-3 beta protein, which plays a role in tumour growth and suppression of the immune system, and may work by re-engaging the immune system to fight cancer
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