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retatrutide cannot be prescribed on the NHS or legitimately via private prescription as it remains unlicensed and without EAMS designation
Both have shown promising results, but they have distinct characteristics and mechanisms that are worth understanding
Although GIP receptor activation may exert anti-emetic effects through brainstem modulation, these effects appear insufficient to completely counterbalance GLP-1 mediated emesis during early treatment and dose escalation ( In contrast, glucagon receptor activation is independently associated with nausea and vomiting through both central and peripheral mechanisms, including effects on gastric motility and autonomic signaling
Metabolic health also changes during this period
