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A GLP-1R agonist (liraglutide) increased endothelial nitric oxide synthase phosphorylation and nitric oxide (NO) production by the 5-AMP-activated protein kinase (AMPK)-dependent pathway and subsequent NO production in cultured HCAEC [56],[57]

Among potential candidates, fingolimod, a sphingosine-1-phosphate receptor (S1P) modulator that prevents lymphocyte egress from the lymph nodes, has shown encouraging outcomes in preclinical and early clinical studies [340]

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Indeed, the crude HR for death (with MOF as a censoring event) in the GLP-1 RA group with the DPP-4i group as reference was 0.94 [0.771.15]