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Simultneamente, se ha demostrado que el complejo GHK-Cu interacta in vitro con componentes del eje de sealizacin de la superfamilia TGF- y regula al alza la expresin de genes anti-apoptticos y de remodelacin de matriz extracelular (ECM), incluyendo aquellos que codifican fibronectina, colgeno I y diversas metaloproteinasas de matriz (MMPs) as como sus inhibidores (TIMPs)
American Journal of Epidemiology
The biggest drawback of BPC-157 is that there is not enough evidence of its safety
Rheumatology (2019) 58(3):42731
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