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Notably, metformins consistent performance across both the DM2AD comorbidity network, where metabolic dysfunction predominates, and the MCI network, where neurodegeneration is central, emphasizes the role of metabolic dysfunction in AD pathophysiology, and highlights metformins promise as a therapeutic candidate for AD prevention in DM2 patients
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BPC-157 has significant anti-inflammatory properties and protects against oxidative stress, aiding in faster recovery from muscle strains, joint damage, and other connective tissue injuries
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