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[7] [8] This triple-receptor mechanism distinguishes it from existing approved agents such as semaglutide (a GLP-1 agonist) or tirzepatide (a dual GLP-1/GIP agonist)

Thus, it prevents the activation of pain and migraine networks in the brain, including disruption of CGRP activity , which has become a central focus in most of the new migraine medications on the market such as the gepants and CGRP monoclonal antibodies

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