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This is proposed to be mediated by TRX1 oxidation by hydrogen peroxide which causes dissociation of ASK1 thereby triggering anti-apoptotic factors via downstream activation of MAPK kinase MKK3/MKK6/p38 and MKK4/MKK7/JNK pathways (Wagner et al
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[1] GHK-Cu acts as a potent attractant for various cells involved in the healing process, including capillary cells, mast cells and macrophages
2018;2:e00034
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