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[2] suggested that BPC-157 may assist repair by supporting tendon fibroblast migration and spreading in laboratory studies, with researchers observing that "F-actin formation as detected by FITC-phalloidin staining was induced in BPC 157-exposed cells," [2] with activation of focal adhesion signaling through phosphorylation of FAK and paxillin proposed to assist cell attachment and movement within the extracellular matrix in these settings

[1] [2] Clinical trials have demonstrated robust efficacy, with A1C reductions of 1.9% to 2.4% and weight loss of approximately 15% at 5 mg and 20-21% at 10-15 mg of baseline body weight in the SURMOUNT-1 trial

They avoid resistance training

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