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TB-500 promotes endothelial cell migration and vessel formation through its actin-regulation mechanism
In wound-healing research, GHK-Cu has been shown to regulate MMP-2 expression and its tissue inhibitors, TIMP-1 and TIMP-2, in fibroblast cultures, indicating a role in controlling ECM remodeling during tissue repair [4]
5B, histological analyses showed that the collagen fibers were arranged in a disorderly manner in the PBS group, but NAC treatment preserved the continuity and orientation of collagen fibers in the NAC group
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