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elegans , GPCR phosphorylation is presumably carried out by grk1 and grk2 (which may work redundantly, as a mutation in grk2 alone does not lead to reduced adaptation nor to hypersensitivity
Their mechanisms and evidence bases are distinct
Researchers are interested in them because some appear to modulate the same GABA, serotonin, and stress-hormone pathways that current anxiety medications target, according to research summarized by the National Institutes of Health
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