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doi: 10.1039/c7fo00809k, 76 MaherP
While dysfunctional oligodendrocytes may exacerbate neurodegeneration, they also exert protective effects, such as secreting the iron-storage protein FTH1 to counter iron-mediated cytotoxicity (Li M
This combination is not typically paired with pramlintide, since cagrilintide already covers amylin signaling and pramlintide is a short-acting amylin analog without new pathway coverage
The treatment requires monitoring by healthcare professionals to ensure safety and effectiveness [1]
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