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What emerges is a coherent biological framework: estrogen maintains the mitochondrial infrastructure and synaptic stability that neurons depend on for sustained function, while GLP-1 receptor activation reduces the inflammatory burden that progressively degrades that infrastructure
Andrew Stokes, associate professor of global health, and a team of researchers from the School of Public Health will seek clarity on the population health effects of GLP-1 drugs with a new grant from the Robert Wood Johnson Foundation
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