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We further demonstrate that oxidative mitochondrial signalling can be used as a biomarker of response to fluoropyrimidine-based chemotherapy treatment in patients with CRC and may have clinical utility for stratifying patients at higher risk who could benefit from metabolically targeted combination therapies
Studies using animal wound models have demonstrated that GHK-Cu accelerates wound contraction, stimulates collagen deposition, and promotes angiogenesis the formation of new blood vessels in damaged tissue
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