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MOTS-c is an exercise-induced mitochondrial-encoded regulator of age-dependent physical decline and muscle homeostasis

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This concentration-dependent effect is epitomized in energy metabolism during exercise, where a shift from white adipose tissue to intramuscular triglycerides as the primary fatty acid source occurs as blood lactate rises from 2 mM to 5 mM, demonstrating lactates role as a metabolic rheostat in real-time [148]

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