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Critically, these effects were achieved without food intake suppression, distinguishing MOTS-c from GLP-1 receptor agonists whose primary mechanism involves appetite reduction
Unlike IGF-1 DES, which benefits from site-specific injection, LR3's long half-life means injection site matters less, the peptide will circulate systemically regardless of where you inject it
[1] [2] Both substances influence metabolic pathways related to glucose regulation and weight management, but through different mechanisms
Initial adjustment period
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