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Presented for research literature review only Hayes JD et al
- Hayley V
Mitochondrial ROS production and oxidative mtDNA damage is increased during reoxygenation in the GPX1 knockout mice, in addition to structural abnormalities in cardiac mitochondria and myocytes, suggesting GPX1 may play an important role in protecting cardiac mitochondria from reoxygenation damage in vivo
They are complementary rather than competing, with BPC-157 addressing structural repair and LL-37 addressing infection defense and innate immune signaling during wound healing
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