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Antioxidant supplements promote tumor formation and growth and confer drug resistance in hepatocellular carcinoma by reducing intracellular ROS and induction of TMBIM1

This imbalance leads to the infiltration of M1 macrophages, forming CD11c+ crown-like structures (CLS) around dead cells in WAT, resulting in the accumulation of high amounts of lipids within macrophages and promoting inflammation [99, 101]

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