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In conclusion, our structural and functional analyses provide a large body of novel information for defining the molecular mechanism underlying the activation of c-MET in response to HGF and NK1 and paves the way for the eventual therapeutic intervention of diseases caused by aberrant activation of c-MET
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los datos disponibles provienen de sistemas in vitro y modelos animales
It may support the bodys ability to recover in selected cases, but it is not a substitute for load management, rehabilitation, and addressing the underlying drivers of the problem
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