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Developed initially for the treatment of type 2 diabetes, semaglutide works by mimicking the effects of the naturally occurring hormone GLP-1 (glucagon-like peptide-1)

Opening of the VGCCs evoked by membrane depolarisation, produced by increasing extracellular K + ([K + ] o ) to 70 mmol/l, stimulated glucagon secretion by over threefold, and this response was reduced by 60% by GLP-1(936) (Fig