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Ramzan F, et al
Engaging all three pathways simultaneously drives glucose-dependent insulin secretion through the two incretin arms while adding glucagon-receptor agonism implicated in energy expenditure, the pharmacological rationale that distinguishes it from single (GLP-1 (Sema)) and dual (GLP-2 (Triz)) incretin agonists
This schedule is just one possible example, not a strict rule
the long-term body-composition outcomes rest more on user reports than on large human trials
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