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It is recognized for supporting energy metabolism and athletic performance due to its role in fatty acid transport and muscle recovery
The first step of the mechanism involves formation of an "acyl-phosphate intermediate"
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This residue is a leucine in IR and a phenylalanine in IGF1R, and we have found that the replacement of leucine-973 in the juxtamembrane region of IR with Phe decreases IRS-1 binding and increases Shc binding
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