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17-AAG (tanespimycin) alleviates disease manifestations in SLE mouse models by reducing inflammation and immune cell activation

It works through multiple mechanisms, increasing blood vessel formation around follicles, reducing inflammation, stimulating collagen synthesis in the dermal papilla, and blocking 5-alpha reductase, the enzyme that converts testosterone to the follicle-damaging DHT

p =0.0011), the increase in baseline PS exposure was significantly dampened by the addition of Albumax (T 1h =0.1% and T 48h =0.7%

Both decisions came after the manufacturers confirmed supply capacity could meet national demand