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This is due to the liver taking up all the circulating FFAs, which then interfere with insulin and IGF-1 signalling (as described above) and eventually leading to hepatic steatosis
These fibres resist digestion in the small intestine and reach the colon, where they are fermented by beneficial bacteria to produce SCFAs that stimulate GLP-1 release
This can happen in malabsorption syndromes, chronic alcoholism, and some neurological issues
More structural improvementslike tendon or ligament integrity, joint function, and enduranceoften develop over 812 weeks as collagen architecture and circulation improve
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