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All clinical endpoints were defined according to Academic Research Consortium-2 (ARC-2) criteria [15]
While mitochondria produce ROS during oxidative phosphorylation, the ROS are appropriately eliminated by several antioxidants under normal physiological conditions
The continuous production of ROS forces the body to consume excess host energy just to survive the biochemical onslaught, directly contributing to the systemic, debilitating fatigue that characterizes these conditions
This creates a frustrating plateau where the medication is neither delivering results nor remaining tolerable
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