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In recent years, studies have revealed that cell death plays a critical role in EMS lesions

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Infiltration of M1-type macrophages into visceral fat leads to chronic release of pro-inflammatory cytokines, such as IL-1, which drive ovarian interstitial fibrosis and hemodynamic changes (62, 63)

Those non-covalent compounds are highly specific and since they bind reversibly, they may exhibit a better safety profile ( in vivo activity (Zhuang et al., 2017), potentially because they are highly polar and hardly able to penetrate the BBB