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Studies have shown [11]: Prevention of gastric lesions when co-administered with NSAIDs Healing of existing NSAID-induced damage Protection of both upper and lower GI tract Preservation of intestinal permeability This is particularly significant given that millions of people take NSAIDs regularly, often experiencing GI side effects
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CYP2C9 genotyping was performed to account for any variability in the pharmacokinetics and pharmacodynamics of CYP2C9-metabolised S-warfarin
Steven et al (134) demonstrated that GLP-1RAs activated GLP-1Rs on platelets via the AMP/PKA pathway, inhibiting sepsis-induced microvascular thrombosis, serving an important role in preventing systemic inflammation and vascular dysfunction, and protecting against organ injury
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