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Through conjugation reactions, glutathione binds to toxins, drugs, and metabolic byproducts, transforming them from fat-soluble compounds that accumulate in tissues into water-soluble forms that can be easily eliminated through urine and bile
The accumulation of LCACs in tumor tissues has been shown to alter the lipid uptake and metabolism of infiltrating T cells, which in turn affects T cell states such as cell division, apoptosis, and cell senescence
The interaction of PD-1 with its two ligands, B7-H1 and B7-DC (PD-L1 and PD-L2), occurs predominantly in peripheral tissues including the tumor microenvironment and leads to apoptosis and downregulation of T-cell effector function [32]
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