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Read the full study: The effects of human GH and its lipolytic fragment (AOD9604) on lipid metabolism following chronic treatment in obese mice and 3-AR knock-out mice (Endocrinology 2001)
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In receptor-deficient systems, the peptide does not produce measurable changes in lipolytic signaling, supporting the conclusion that its effects are mediated through adrenergic pathway engagement rather than direct hormone receptor activation
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