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Type IV pili of pathogenic Neisseriae elicit cortical plaque formation in epithelial cells

Abstract Glucose-6-phosphate dehydrogenase (G6PD) deficiency, one of the most common human genetic enzymopathies, is caused by over 160 different point mutations and contributes to the severity of many acute and chronic diseases associated with oxidative stress, including hemolytic anemia and bilirubin-induced neurological damage particularly in newborns

BMC Plant Biol 24:866

Collectively, these microbiota-modulating therapies offer a diverse therapeutic entry point capable of changing metabolic, immunological, and neurotrophic pathways involved in PD development [167, 168]